8/19/2011

Left Ventricular Hypertrophy: Physiology versus Pathology (Developments in Cardiovascular Medicine)

Left Ventricular Hypertrophy: Physiology versus Pathology (Developments in Cardiovascular Medicine)


ventricular wall - click on the image below for more information.



ventricular wall


The importance of left ventricular hypertrophy (LVH) in cardiovascular disease has gained wide recognition. LVH is a highly important risk factor associated with major cardiovascular events, including symptomatic heart failure, particularly in patients with systemic hypertension. In recent years much has been learned about the genetics, molecular background, prevalence, incidence and prognosis of LVH. A variety of noninvasive methods has emerged for detecting LVH and the assessment of rev





Left Ventricular Hypertrophy: Physiology versus Pathology (Developments in Cardiovascular Medicine)





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Comparing Atrial and Ventricular Septal Defects

Article by Josephine Osborn










Septal defect is a malformation in the heart characterized by a hole in the wall separating the left and right sides. It is a congenital heart defect and has two kinds, the atrial septal defect and the ventricular septal defect. The kind of septal defect depends on the location of the hole. In atrial septal defect, a hole is found in the wall (septum) between the left and right upper chambers (atria) of the heart. A hole in the wall of the lower chambers (ventricle) of the heart characterizes ventricular septal defect. Various factors are believed to cause this condition; some of them are genetics and side effects of drug, like Paxil side effects, taken by mother during pregnancy.

About 9 percent of all congenital heart defects is atrial septal defect. The location of the hole determines the 3 sub-classifications of the condition. The size and position of the opening determines the symptoms a child manifests. Generally, the condition is asymptomatic wherein children feels well, grows and gains weight normally just like any child. But with a larger hole and a more severe ASD, a child may experience poor weight gain, decreased exercise tolerance, frequent upper respiratory tract infections, poor growth, shortness of breath and poor appetite. ASDs are not always diagnosed early in life. The heart murmurs of ASD are not as audible as the other murmurs making it harder to determine and be diagnosed sometime between infancy and adolescence. Grave complications may result if the condition stays untreated while the child is growing. Fortunately, ASD may be discovered even before the onset of symptoms. Cardiologist suggests that closure of the hole be done as soon as possible.

Ventricular septal defects, the most common type of CHD, account for approximately 25% of all congenital heart defects. The severity of the symptoms depends on the size of the hole. Small VSDs produce no symptoms and has high spontaneous closure rate during the first year of life. It can be determined by the health care provider when a loud heart murmur is audible upon auscultation. Symptoms of large VSDs begin to manifest 1-6 months after birth when the left ventricle begins to fail. Feeding difficulties, poor weight gain, failure to thrive, very fast heartbeats, sweating, fast breathing, and pallor are some of its symptoms. If it remains undetected and untreated, it can cause more serious health problems. If not repaired surgically, it may lead to irreversible pulmonary hypertension, which has a poor prognosis.

Both atrial and ventricular defects are fatal. The key to this, is early detection and management. It is best to keep a regular check-up with a health care provider. If you want to know more about this condition and other birth defects, please see your health care provider.



About the Author

Paxil is believed to cause many birth defects together with congenital heart disease among newborns of ladies who took this drug throughout pregnancy. Apprehend a lot of regarding the "Paxil side effects" and also the drug's aspect effects here.













Orignal From: Left Ventricular Hypertrophy: Physiology versus Pathology (Developments in Cardiovascular Medicine)

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